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A more likely mechanism then is that Stats control the transcription of other genes that participate in the stabilization of the myc mRNA. The control of the stability of short-lived mRNAs such as myc is under intense scrutiny, but how mitogenic signals impact this control is not yet fully understood Ross, ; Guhaniyogi and Brewer, The Tyr , site is also a canonical site for Grb2 binding. The microinjection approach used for these studies precluded a more detailed analysis of how SFKs affected Myc production. Ligand-induced dimerization of the EGF-R results in the rapid phosphorylation of various EGF-R tyrosine residues, including positions , , , , and Wright et al.
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The interplay between Src family kinases and receptor tyrosine kinases

Other studies have implicated the SFK-mediated tyrosine phosphorylation of dynamin in the control of endocytosis Ahn et al. The control of the stability of short-lived mRNAs such as myc is under intense scrutiny, but how mitogenic signals impact this control is not yet fully understood Ross, ; Guhaniyogi and Brewer, Thank you for visiting nature. Oncogene 23, — doi Signaling is initiated by the recruitment of proteins such as Shc and Shp2 to the activated receptor. Cell , 11 , —
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The data suggested a model in which Vav2 is phosphorylated by SFKs in response to mitogen stimulation, thus initiating the activation of a Rac-dependent pathway that controls the expression of the myc gene. While an absolute requirement for SFKs may depend on the cell type and the cellular context under study, the available data suggest that SFKs promote mitogenesis from a variety of RTKs. In the meantime, to ensure continued support, we are displaying the site without styles and JavaScript. In summary, we have learned much in recent years about how SFKs are regulated by, regulate, and participate in signaling from, RTKs. Here care must be taken that the inhibitor has sufficient selectivity. In fibroblasts, early reports suggested that the basal transcription of the myc gene was detectable in unstimulated cells, and unaffected by mitogen treatment, rather mitogen stimulation acted to stabilize myc mRNAs once made Blanchard et al. The control of the stability of short-lived mRNAs such as myc is under intense scrutiny, but how mitogenic signals impact this control is not yet fully understood Ross, ; Guhaniyogi and Brewer,
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Antimicrobial Agents and Chemotherapy Blood , 94 , — It is well established that both the introduction of activated Src into cells and the stimulation of cells with peptide mitogens such as PDGF lead to rapid and profound changes in the cytoskeleton. Whether Ras signaling within the Golgi participates in mitogenesis is not clear. Here care must be taken that the inhibitor has sufficient selectivity. The interplay between Src family kinases and receptor tyrosine kinases.
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